Author/Editor		Kanič, Z; Todorovič-Guid, M; Roškar, Z; Pogorevc, R; Bračič, K
Title		Hipoksična okvara miokarda asfiksičnega novorojenčka
Translated title		Hypoxic myocardial injury in the asphyxiated newborn
Type		članek
Source		In: Gregorič A, editor. Nutritivna alergija. Farmakologija perinatalnega obdobja. Skrb za zdravje v prvem letu življenja. Zbornik predavanj 13. srečanje pediatrov v Mariboru z mednarodno udeležbo; 2003 apr 4-5; Maribor. Maribor: Splošna bolnišnica Maribor,
Publication year		2003
Volume		str. 141-4
Language		slo
Abstract		Perinatal asphyxia is a frequent aetiological factor in the damage to the heart and other organs after birth. High- risk newborns should be assessed for the degree of asphyxia. In asphyxiated newborns the brain, kidney, heart, gut and lungs are the most involved organs. Hypoxic myocardial injury can be assessed clinically, electrocardiographically, echocardiographically, by chest x-ray and enzymatically: lactate dehydrogenase (LDH), creatinine kinase (CK) and troponin T. Heart injury may consist of transient myocardial ischaemia, cardiac decompensation or myocardial necrosis. Continuous re-evaluation and treatment of cardiac decompensation is necessary Cardiac Troponin T is an ideal marker for myocardial necrosis. It is a myocardial muscle specific protein. Normal values in cord blood and in the serum of newborns and premature babies have not yet been defined. In comparison with the brain, the heart is less often damaged in perinatal asphyxia. Clinical re-evaluation and further examinations enable the identification of myocardial damage, which can sometimes masquerade as respiratory distress syndrome. Treatment usually results in rapid improvement with no residual morbidity.
Descriptors		ASPHYXIA NEONATORUM MYOCARDIAL ISCHEMIA INFANT, NEWBORN